Which statement describes a secondary effect of angiotensin II?

Which statement describes a secondary effect of angiotensin II?

• A. Decreases aldosterone secretion
• B. Increases aldosterone secretion
• C. Causes bronchodilation
• D. Inhibits vasoconstriction

Answer: (B)

Angiotensin II has several well-known downstream effects that help raise blood pressure and preserve fluid balance. A secondary effect of Ang II is stimulating the adrenal cortex to secrete aldosterone. This increases aldosterone levels, which act on the kidney to reabsorb sodium (and water follows), expanding blood volume and contributing to higher blood pressure. This hormonal cascade—Ang II driving aldosterone release downstream of the renin-angiotensin system—is a key part of how the body fine-tunes fluid balance and vascular tone. So the statement describing an increase in aldosterone secretion best captures a secondary effect of Ang II. The other options describe actions that aren’t characteristic secondary effects: Ang II does not decrease aldosterone (it stimulates it), it does not cause bronchodilation as a noted effect, and it does not inhibit vasoconstriction (it promotes vasoconstriction).

Angiotensin II has several well-known downstream effects that help raise blood pressure and preserve fluid balance. A secondary effect of Ang II is stimulating the adrenal cortex to secrete aldosterone. This increases aldosterone levels, which act on the kidney to reabsorb sodium (and water follows), expanding blood volume and contributing to higher blood pressure. This hormonal cascade—Ang II driving aldosterone release downstream of the renin-angiotensin system—is a key part of how the body fine-tunes fluid balance and vascular tone.

So the statement describing an increase in aldosterone secretion best captures a secondary effect of Ang II. The other options describe actions that aren’t characteristic secondary effects: Ang II does not decrease aldosterone (it stimulates it), it does not cause bronchodilation as a noted effect, and it does not inhibit vasoconstriction (it promotes vasoconstriction).